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If GHK-Cu were developed as a pharmaceutical drug, its applications would likely be limited to specialized medical fields, such as wound healing and injectable therapies

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Thus, damage to mitochondrial DNA and loss of can further increase ROS production, thereby creating a cycle of dysfunction that includes increased ROS production by the respiratory chain, activation of signal transduction pathways, and potentially, leading to apoptosis or necrosis, as well as the release of cytochrome c, which triggers apoptosis

(visit source) Zandi PP, Anthony JC, Khachaturian AS, et al