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doi: 10.1016/j.drudis.2014.10.003

They have also identified a plethora of pathological molecular and neuronal phenotypes observed in schizophrenia patients, confirming the translational significance of these pre-clinical models [6, 7]

For example, putative anti-apoptotic smoking-induced effects on Bcl-2 or p53 lead to a higher risk for malignancies and might lead to unbalanced drop-out in the smoking group

BPC 157 likely counteracted Virchow's triad, as it did in rats with infrarenal inferior vena cava occlusion[21]

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