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By regulating neuroinflammation, oxidative stress, and abnormal protein clearance, they directly or indirectly activate multiple cell death pathways, driving dopaminergic neurodegeneration [238] (Fig

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[DOI] [PubMed] [Google Scholar] 34.Fu C, Liang Q, Ma L, et al

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The -synuclein that has accumulated interacts to the proteasome and effectively suppresses proteasomal action
