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Nuclear mitochondrial gene abnormalities were ruled-out using an oligonucleotide array with comparative genomic hybridization analysis that examines 180 nuclear genes involved in mitochondria function, including genes involved in fatty-acid oxidation, carnitine metabolism, mitochondrial biogenesis, mtDNA maintenance, transcription and translation, and ETC complex assembly (MitoMet, Baylor Medical Genetics Laboratory)

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or in non-cancerous conditions where there is upregulation of arginase [65], including sepsis where there are reduced L-Arg levels and excessive protein catabolism [93]

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Additionally, they also determined that the expression of protein kinase CPG16 increased, pointing to its importance in suppressing insulin gene expression in pancreatic beta cells under cytotoxicity (164)