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doi: 10.3390/cells9112446
By constructing and analyzing networks of genes, we can elucidate the regulatory circuits that drive cancer progression
How autophagy can restore proteostasis defects in multiple diseases
Targeting metabolic vulnerabilities in specific macrophage subsets, such as limiting ferroportin-mediated iron export in M2 TAMs or enhancing ACSL4-dependent lipid peroxidation in inflammatory TAMs, may shift the TME toward a ferroptosis-permissive state
This minimizes any potential interaction
