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Both the acid and the amide are precursors to the coenzyme nicotinamide adenine dinucleotide

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Due to its elimination of neurological protective effects against H 2 O 2 and elevation of thioredoxin-1 by suppressing the nuclear factor erythroid 2-related factor 2 signal pathway, mutant DJ-1 (L166P and M26I) raises the vulnerability of SH-SY5Y cells to oxidative stress [211]

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