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These results may explain, in part, the finding that low-dose dietary zinc supplementation extended the lifespan of SOD1 G39A transgenic mice (Ermilova et al., 2005), as this mutation is known to reduce Sod1s affinity for zinc (Crow et al., 1997)

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Patients with idiopathic dilated cardiomyopathy exhibit elevated copper but reduced serum zinc compared to healthy controls, supporting previous findings linking zinc deficiency to cardiac health [82]