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The imbalance between Th1, Th17, and Treg cells exacerbates the inflammatory environment

Pharmacokinetics of liothyronine and levothyroxine

ROS-induced mitochondrial DNA damage impairs mitochondrial function, promotes apoptosis of vascular smooth muscle cells and macrophages, inhibits smooth muscle cell proliferation, increases necrotic core size, reduces fibrous cap area, and increases plaque vulnerability

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1J) in the presence of ferrostatin-1 (an inhibitor of ferroptosis), due to the higher sensitivity of T47D to GPX4 loss, which was highly sensitive to palbociclib when ferrostatin-1 was removed (Fig
