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doi: 10.1016/j.ajpath.2014.10.009 134 Veses-GarciaM.LiuX.RigdenD

In the context of periodontitis, the increased expression of SLC7A11 likely represents a compensatory response to elevated oxidative stress, as ferroptosis characterized by iron-dependent lipid peroxidation has been shown to be involved in inflammatory diseases including periodontitis [24]

The median PFS of the tepotinib group and sorafenib group was 2.8 and 1.4 months, respectively, and the median overall survival was 9.3 and 8.6 months, respectively

For example, although the relationship between P53 and ferroptosis has been well established[88, 211, 212], there is no evidence suggesting that its methylation is involved in the regulatory effect of P53 on ferroptosis

As discussed below, such domains will spontaneously aggregate together with similar or different proteins, increasingly destabilizing the function of such oxidatively modified proteins and eventually leading to cell death