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XPF silencing increases cisplatin induced cell death Since a higher amount of DNA damage, as shown by the H2AX analysis, correlated with increased cell death, we aimed to explore whether increased DNA repair capacity is responsible for A549 cisplatin resistance phenotype

When mixed, it may cause unwanted reactions and side effects

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Since the NADPH oxidase-mediated respiratory burst is a major microbicidal mechanism of neutrophils (21, 56) and a diminished respiratory burst activity was seen in C

Furthermore, such heterotypic interactions do not necessitate classical -sheet-rich amyloid fibril formation, but rather favor amorphous, lipidrich, and membranedense inclusions