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found that IRAK3 (IRAK-M) is markedly upregulated in sepsis, particularly in alveolar macrophages, where it suppresses TLR signaling and reduces cytokine production
Lung tissue samples were taken during the necropsy and put in a 10% buffered formalin solution for storage
Nada MA, Chace DH, Sprecher H, Roe CR
Arch Intern Med 1999;159:1143-4
