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Mechanistically, EGCG promotes mitochondrial dysfunction and ROS accumulation in hepatocytes, depleting intracellular glutathione and triggering apoptosis via caspase-3 activation
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Additionally, novel compounds such as the SERMs/SERDs hybrid bazedoxifene and the selective ER covalent antagonists are undergoing phase 2 clinical trials for patients experiencing disease progression following advanced endocrine therapy
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