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Pore, D

ATF3 can activate transcription and expression of RNF146, thereby inhibiting OGD/R-induced neuronal ferroptosis

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Oxidative stress and neuroinflammation are increasingly recognized as central elements in its development, with mitochondrial dysfunction and weakened antioxidant systems playing a significant role

Mechanistically, -Syn oligomers interact with mitochondrial respiratory chain complex I, impairing its function, leading to mitochondrial membrane potential depolarization, and promoting the release of proapoptotic factors into the cytoplasm