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The mechanism of pathophysiological action of Lp(a) associated with CVD risk involves multiple pathways, most notably an apoB-mediated proatherogenic effect [27, 28] and a pro-inflammatory effect through its oxidised phospholipid content [2931]

doi: 10.1007/s13238-019-0612-5 311 ZhouX

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In fact, we have learned that the biogenesis and regression of LDs are driven by complex mechanisms that are coupled to lipid homeostasis

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