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This change in the binding affinity of NBD-Sph to the NTD of NCR1 could be caused by an altered hydrogen binding capacity of the sphingosine backbone when the pH changes, as has been shown to take place for natural sphingosine (47)

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By locking microtubules into a rigid, non-dynamic state, these agents interfere with mitochondrial trafficking, vesicle transport, and axonal signaling, culminating in intra-axonal congestion and distal axon degeneration [13]

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[DOI] [PubMed] [Google Scholar] 8.Loo V.G., Bourgault A.M., Poirier L., Lamothe F., Michaud S., Turgeon N., Toye B., Beaudoin A., Frost E.H., Gilca R., et al