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MD simulations also identified a natural compound schisandrin B (Sch B) that specifically bound to the ligand-binding domain of PXR, induced conformational changes and disrupted the PARP1PXR interaction interface, thus suppressed the ribosylation

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GHK-Cu also enhances the production of specialized pro-resolving mediators (SPMs), including resolvins and protectins, which actively promote the clearance of inflammatory debris and the restoration of tissue homeostasis

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