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The rationale is: First course: Addresses the existing senescent cell burden
Lapatinib induces mitochondrial dysfunction to enhance oxidative stress and ferroptosis in doxorubicin-induced cardiomyocytes via inhibition of PI3K/AKT signaling pathway
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A lightweight serum formula that absorbs quickly and leaves no greasy residue or white film
Forty-five patients (8.5%
