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Mori H, Kondo T, Yokochi M, Matsumine H, Nakagawa-Hattori Y, Miyake T, Suda K, Mizuno Y (1998) Pathologic and biochemical studies of juvenile parkinsonism linked to chromosome 6q

This could be an important natural element to protect mitochondria from electron leakage, mito-stress, succinate accumulation and thus reduce HIF-1 activation

This process is primarily catalyzed by three types of enzymes, also known as m6A regulators, including methylases (writers), demethylases (erasers) and m6A-binding proteins (readers), making m6A modification dynamic and reversible [78]

A.BellamyL.PariseG.BakerS

and Mullin S., The Genetics of Parkinson's Disease and Implications for Clinical Practice, Genes 12, no
