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A recent study suggests that rotenone administration activates caspase-2 in mice neurons inducing the activation of downstream apoptotic effectors such as Bid, Bax, caspase 3 and 9, thus initiating apoptosis [67]

Notably, POR's ubiquitous presence in various cancer cell lines suggests its potential significance in lipid peroxidation and ferroptosis [158, 159]

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