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Description
The results suggest that HTYs in vivo cardioprotective effects may be mediated in part by its circulating metabolites rather than the parent molecule, therefore, the demonstrated signaling modulation of free HTY effects should be interpreted as indirect modulation through oxidative stress-related markers rather than direct plaque-regressive action

Results and data analysis Figures 1 and 2 show the findings of spectrum measurements in iron-mediated oxyhemoglobin oxidation on hemolysate with regard to time in minutes

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