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At the same time, more of your existing NAD+ gets consumed by enzymes like CD38 and PARPs, which are activated by things like inflammation, oxidative stress, and DNA damage

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The knockout mouse data is particularly useful for interpreting this: long-term AOD-9604 treatment in beta3-AR-deficient mice failed to produce body weight reduction or sustained lipolysis, confirming that the chronic fat loss effect depends on this receptor pathway

Synergy is inferred from the individual mechanisms, not demonstrated in combination

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