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Simultaneously, it activates extracellular signal-regulated kinase (ERK1/2) pathways while reducing pro-inflammatory nuclear factor kappa B (NF-B) signaling a combination that supports tissue recovery while managing inflammation

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Metabolome analysis using capillary electrophoresis time-of-flight mass spectrometry (CE-TOFMS) in combination with [ 13 C]-labeled glycolytic metabolites provided strong evidence that glucopiericidin A suppresses glycolysis by functionally targeting GLUT1-mediated glucose uptake [60]

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