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[DOI] [PMC free article] [PubMed] [Google Scholar] 20.Ten W., Tseng C.-C., Chiang Y.-S., Wu C.-L., Chen H.-C

This potential restoration of p53s function by the FOXO4-DRI peptide might enable it to trigger the programmed cell death of senescent cells, essentially clearing out cells that might otherwise contribute to the decline in tissue function seen in aged cell cultures

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Exogenous sources also contribute significantly to free radical formation [3]