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In addition to its pro-differentiating actions, nicotinamide promotes neuronal survival, especially in oxidative stress environments, through various pathways, such as maintenance of protein kinase B (akt)-dependent phosphorylation of forkhead transcription factor (FOXO3a)
Furthermore, the construction of mathematical frameworks such as this can be implemented to explore other classes of compounds and mechanisms of toxicity as a function of their physicochemical properties, while providing an alternative method of quantifying experimentally elusive radical species
