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Addressing this gap is critical, as personalized probiotic strategies could optimize therapeutic benefits while mitigating risks in SLE management ( 5.4 Dosage, survival, and delivery systems of probiotics Most investigations of probiotic effectiveness in SLE underscore the importance of optimum dose maximization for immune modification

Mitochondrial dysfunction in neurodegenerative diseases: a focus on iPSC-derived neuronal models

by restoring some leptin responsiveness, an amylin analog may lower that defended set point and make the appetite suppression from GLP-1 agonism deeper and more durable

No NETs no TIME: crosstalk between neutrophil extracellular traps and the tumor immune microenvironment

Nucl Recept Signal 2008
