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17-hydroxyprogesterone levels may be necessary to rule out an adrenal cause of androgen excess

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La Merie Publishing Blockbuster Biologics 2012 2013 202

Crucially, in all three cell lines, the de novo GSH synthesis inhibitor buthionine sulfoximine (BSO) abolished the ability of MPA and Pyr pretreatment to inhibit ferroptosis, consistent with a GSH-dependent protective mechanism (Fig 4B)

Impaired kidney function is also associated with increased IGFBP levels (e.g., IGFBP-1, -2, -3, -4, and -6) (75, 77) and IGFBP-3 protease activity, which subsequently increases low-molecular-weight IGFBP-3 fragments (78), most likely due to a compromised renal IGFBP-clearance ( Figure 4 )