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Nanomedicine 12 , 655664 (2016)

(2021) reported that m6A modification appeared to enhance Beclin-1-mediated autophagy and consequently trigger ferroptosis in human HSCs, which is further confirmed by silencing YT521-B homology domain family, a critical m6A reader protein for sustaining Beclin-1 mRNA stability

doi: 10.1146/annurev.med.52.1.503 25 KeefeR

Matching treatment intensity to disease biology is critical to achieving a high therapeutic index which maximizes oncologic control and minimizes normal tissue toxicity

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