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187 WatsonJ
Hedman CA, Frystyk J, Lindstrm T et al (2004) Residual beta-cell function more than glycemic control determines abnormalities of the insulin-like growth factor system in type 1 diabetes
doi: 10.1371/journal.pone.0154427
The peptide has also been associated with normalised NO signaling under both excessive and suppressed NO states, counteracting the implications of NOS blockade and NO overproduction
This is remarkable for any pharmaceutical compound and reflects AOD 9604 highly targeted mechanism that affects fat cells without interfering with other physiological systems
