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Microbiol Spectr 10:e02408-e2421

Consistent with an iron-sulfur cluster defect, SLC25A39/40 knock out cells displayed reduced activity of the enzyme aconitase, in which iron sulfur clusters function in both substrate binding and catalysis (Fig 4f)

) promote tyrosine nitration by inducing tyrosine oxidation and also by reacting with NO 2 to produce

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Researchers at Baylor College of Medicine in Houston combined studies of older adults and older mice since both groups were glutathione-deficient, which is a common side effect of aging