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There appears to be an anomalous storage of mitochondrial ATP synthase subunit c that is neither the result of mutation nor enhanced expression of the protein but rather a slower degradation of the mitochondrial ATP synthase in comparison with normal cells [116]

[11] Your prescriber will individualize both the route (IV or IM) and the timetable based on your weight, kidney function, and overall clinical picture

Saunders Co

Acknowledgements Not applicable

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