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Cysteine, due to its limited intracellular availability, is the rate-limiting precursor in GSH synthesis

This review summarizes the findings on the role of oxidative stress and mitochondrial dysfunction in the pathophysiology of AD, with a focus on the role of CoQ 10

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Other promising candidates include the products of the PGE metabolic pathway, which may reflect the activity of inflammatory pain [230]

Vitamin D administration may reduce TPOAb and TgAb titers by improving thyroid function through immunomodulation
