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Endothelium-derived nitric oxide (NO) is a key regulator of microvascular endothelial cell survival and a negative modulator of vascular endothelial growth factor (VEGF) signaling

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Inhibition of miR-192 leads to increased expression of stearoyl-CoA desaturase 1 (SCD-1), the enzyme that catalyzes the biosynthesis of monounsaturated fatty acids [317], resulting in increased TG accumulation