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Any mouse that resisted prodding and remained on the platform for 30 s was considered to be exhausted, and time was recorded
The recessive form of the disease is caused primarily by null variants, although amino acid substitutions, splice junction variants, and missense variants have also been reported
Egf1.0 mutant constructs indicated that Egf1.0 blocks proPO activation via PAP inhibition
10.1111/J.1749-6632.2009.04925.X 37 SoicaC.DanciuC.Savoiu-BalintG.BorcanF.AmbrusR.ZupkoI.et al (2014)
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