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Description
The ataxia telangiectasia mutated (ATM) protein detects DNA double-strand breaks and promotes DNA damage repair
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530 Given the elevated activity of monoamine oxidase-B (MAO-B) observed in AD, dual inhibition of AChE and MAO-B holds promise for synergistic effects on cholinergic system recovery and A plaque formation, along with potential benefits in alleviating oxidative stress injury
Furthermore, SLC7A11 upregulation in A549 cells diminishes PD-L1 expression and suppresses ferroptosis, suggesting an association between ferroptosis and immunotherapeutic outcomes [93]
