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As a humanized monoclonal antibody against NGF, tanezumab has a high affinity and specificity for bonding with NGF, thus blocking the mediator from engaging with nociceptive nerve cell receptors

Choice A: A defect in chromosome 13 is the cause of Patau syndrome (trisomy 13)

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Three mechanistic explanations, not mutually exclusive: Signalling cascade persistence: BPC-157 activates VEGFR2 and FAK-paxillin pathways whose downstream effects (angiogenesis, fibroblast migration, collagen remodelling) unfold over days to weeks after initial receptor engagement