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2000 Aug 4;87(3):214-20
This distinction is clinically important: oral estrogens trigger a hepatic response that elevates clotting factors, C-reactive protein, sex hormone-binding globulin, and triglycerides, while transdermal formulations do not produce these effects at standard doses [9]
5, 75 Thus, data in hypertrophied hearts with normal systolic function do not support a model that impaired ATP-producing capacity might be an antecedent to the transition to decompensated HF
A few cases in small children with PCD showed that liver size and enzymes were normalized after treatment with L-carnitine [52,56,57] 3.3
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