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However, under oxidative stress or pathological conditions like AGEs (72), Nrf2 is activated and translocated to the nucleus, initiating the expression of antioxidant genes such as GPX4, which are essential for reducing lipid peroxidation and delaying ferroptosis (73)

Research has shown these genes can be turned on via cell-signaling pathwaysthe major one being Nrf2

It helps regenerate oxidized glutathione, thereby supporting its function as a potent antioxidant and detoxifier

Thiram-induced cytotoxicity is accompanied by a rapid and drastic oxidation of reduced glutathione with consecutive lipid peroxidation and cell death

[DOI] [PMC free article] [PubMed] [Google Scholar] 36.Roberto O.Y.-J., Ivan M.Q.-D., Daniel A.-A., Kelly S.-M., Albert V.-G., Jazminy M.-G., Ronald Y.-M., Edmundo A.V.-C., Rafael J.-A., Pedro Buc C., et al