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Our hypothesis calls for precision nutrition models that stratify patients based on biochemical profiles and sex-specific responsesgenerating new paradigms for thyroid-CVD comanagement [9]

Luck MR, Jeyaseelan I, Scholes RA

doi: 10.1038/s41419-022-05115-x 20 DuJWangTLiYZhouYWangXYuXet al

doi: 10.1007/s10637-005-1444-0

Under physiological conditions, Nrf2 remains sequestered in the cytoplasm by its inhibitor, the Kelch-like ETC-associated protein 1 (Keap1), but oxidative or electrophilic stress triggers Nrf2 dissociation, nuclear translocation, and binding to antioxidant response elements (AREs), upregulating genes such as heme oxygenase-1 (HO-1), NAD(P)H quinone oxidoreductase 1 (NQO1), and glutathione S-transferases (GSTs) (Cuadrado et al., 2019)