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Since the ultimate goal of the project was to produce a clinically relevant molecule that could be delivered peripherally but still exhibit pro-cognitive/anti-dementia activity, the effectiveness of both the intraperitoneal and oral delivery routes of dihexa administration were determined using the scopolamine model
In addition to cell cycle arrest, 1D228 reversed the EMT by upregulation of E-cadherin expression and downregulation of N-cadherin, which significantly blocked the tumor cell migration
Plasticity of Adipose Tissue-Derived Stem Cells and Regulation of Angiogenesis
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