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Impaired cholesterol absorption exacerbates synaptic deficits under low-cholesterol conditions and hinders the increase in synapses under high-cholesterol conditions Because ncr-1(0) mutants exhibited lower levels of cholesterol, we proceeded to investigate whether cholesterol plays a role in the regulation of synapse development mediated by NCR-1

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High density lipoprotein-induced angiogenesis requires the activation of Ras/MAP kinase in human coronary artery endothelial cells

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Development 132 , 41434154

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