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Utilizing proteomics, we identify elevated levels of Pin1 in preclinical AD (PCAD) brain [60], consistent with the notion that PCAD subjects, characterized by normal scores on tests of cognition but having AD-like pathology in brain, respond to elevated A by increasing expression of Pin1

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AMPKalpha1-mediated ZDHHC8 phosphorylation promotes the palmitoylation of SLC7A11 to facilitate ferroptosis resistance in glioblastoma