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Treatment with 2 mM H 2 O 2 completely inhibited the growth of HKU16 gshT , while it only extended the lag phase of the HKU16 wild-type and complemented strains, which both reached bacterial densities equivalent to those of untreated cultures in the stationary phase (Fig
Thus, timing and dosage become critical factors
These changes induce insulin resistance and mitochondrial dysfunction, eventually leading to metabolic disorders and obesity
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