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For two weeks, study subjects were given 0.81 millimoles per kilogram (mmol/kg) cysteine and 1.33 mmol/kg glycine (2)

Increased levels of programmed cell death 5 (PDCD5) subsequently ameliorated progressive fibrosis and cardiac dysfunction by inhibiting histone deacetylase 3 (HDAC3), suggesting that PDCD5 serves as an endogenous negative feedback regulator in progressive cardiac fibrosis [109]

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