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This accelerated resolution of inflammation corresponds with faster progression through healing phases

[6] Release of GST-A1 as an indication of cellular necrosis [edit] Increases in serum and urinary GST-A1 have been found in association with hepatocyte and renal proximal tubular necrosis respectively, and have potential for monitoring injury to these tissues

Moreover, the ex vivo perfusion system is metabolically stable in contrast to the gestational state