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Description
doi: 10.1016/s0167-5273(00)00320-x 102 PekarovaMKubalaLMartiskovaHPapezikovaIKralovaSBaldusSet al

Proal A, Marshall T

In biochemical and preclinical models, the peptide appears to regulate endothelial activity, cell migration, and cytoskeletal organization through interactions with kinase signaling cascades and nitric oxide related mechanisms

Studies in animal models of inflammatory bowel disease (IBD) demonstrate BPC-157 can reduce inflammation, promote mucosal healing, and protect against intestinal damage

Proteolytic inactivation of MAP-kinase-kinase by anthrax lethal factor
