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A significant improvement in basal and maximal mitochondrial respiration in vitro was also observed, suggesting a potential approach for treating AD-related mitochondrial dysfunction via intranasal delivery (Wong and Ho, 2018)

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These structural and functional impairments underscore that SYKs role in SC metabolism is not cell-autonomous but is fundamental to the broader regenerative process by ensuring axons receive the necessary bioenergetic support

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J., Smith, A., Heckelman, P
