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At the single center with 20 years followup, the 35 patients followed had weight loss of 30.1 kg, 48.9% EWL and 22.2% total weight loss

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By integrating metabolomic and proteomic data, we identified both shared and subtype-specific molecular signatures across the two PD subtypes

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Further, these data suggest that at acute time points following Gclc genetic deletion, compensation by NRF1 could be responsible for the dispensable role of NRF2 in GSH-depleted conditions