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Another mechanism that plays an important role in the pathogenesis of COPD is apoptosis and interestingly, increased apoptotic events correlate with ECs damage [103]
The loss of function is often disproportionately large as the result of acute pressure lesions compared with the extent of pathologic change
BDNF-AS expression is increased in early onset but not in late onset AUD amygdala and appears to be regulated epitranscriptomically via decreased N 6-methyladenosine on BDNF-AS
Reactive oxygen species and aging in Caenorhabditis elegans: causal or casual relationship
