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TMAO and endoplasmic reticulum kinase PERK TMAO, a gut microbiota-derived metabolite, has emerged as a critical modulator of ERS through its interaction with the PERK pathway

Supports energy metabolism

Contrastingly, there was a significant increase in the peak inward Ca 2+ current in the presence of 100 M TMAO, which was reversed by washout with the NT solution (Figures 6D,E)

These findings are complemented by the identification of several single-nucleotide polymorphisms (SNP) in GKRP, the protein that inhibits and sequesters GCK during fasting, that strongly associate with lower fasting glucose and insulin resistance coupled with increased blood and liver TAGs (243, 296)

doi: 10.1007/s00018-014-1579-2