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Ingraham NE, Lotfi-Emran S, Thielen BK, Techar K, Morris RS, Holtan SG, et al

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The pathological hallmark of the disease is a protein called as -synuclein that binds to ubiquitin and forms cytoplasmatic inclusions referred to as Lewy bodies which are round eosinophilic intraneuronal filamentous inclusions [114] thereby causing degeneration of the dopaminergic neurons [115]

In addition, treatment with either MCC-950 (NLRP3 inhibitor) or anakinra (IL-1ra) reverses the cardiac electrical remodelling and decreases the incidence of arrhythmias, emphasizing the pathomechanistic relevance of IL-1 and also a novel pharmacological strategy for the treatment of DM-associated arrhythmias